イオン対試薬用ソルナック チューブは2タイプ ★ OOAN(カチオン交換) :トリエチルアミン，ジブチルアミンによるイオン化抑制の改善★ CAOO(アニオン交換)…
Posted: 2021-09-22 19:00:00
Acute lung injury (ALI), a common complication after traumatic brain injury (TBI), can evolve into acute respiratory distress syndrome (ARDS) and has a mortality rate of 30-40%. Secondary ALI after TBI exhibits the following typical pathological features: infiltration of neutrophils into the alveolar and interstitial space, alveolar septal thickening, alveolar edema and hemorrhage. Extracellular vesicles (EVs) were recently identified as key mediators in TBI-induced ALI. Due to their small size and lipid bilayer, they can pass through the disrupted blood-brain barrier (BBB) into the peripheral circulation and deliver their contents, such as genetic material and proteins, to target cells through processes such as fusion, receptor-mediated interactions, and uptake. Acting as messengers, EVs contribute to mediating brain-lung crosstalk after TBI. In this review, we aim to summarize the mechanism of EVs in TBI-induced ALI, which may provide new ideas for clinical treatment. Keywords: Acute Lung Injury; Cogulation; Extracellular vesicle; Inflammation; Traumatic Brain Injury.
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